Publish with Us

Cancer Research and Therapy Connect

Eyad Elkord
Editor-in-Chief

Eyad Elkord
Editor-in-Chief

View Profile

Cancer Research and Therapy Connect is a peer-reviewed, open-access journal dedicated to advancing precision and targeted approaches in cancer treatment. The journal publishes innovative research that deepens understanding of the molecular and immunological mechanisms driving cancer progression and therapeutic response.

Volumes 2
Articles 14
Volume: 2, 2026

Insights

18 Days

Time to First Peer Review Decision

24 Days

Time to Final Acceptance

5 Days

Acceptance to First Online


Recent Articles

open-access Mini-Review

Available Online: 09 Jun 2026

Tumor Suppressor Gene Loss and Immune-Epigenetic Reprogramming

Volume 2

open-access Perspective

Published: 22 May 2026

Chrono-Adaptive Resistance Disruption (CARD): A Novel Pharmacological Framework to Address Anticancer Drug Resistance

Volume 2

Drug resistance in anticancer therapy has long been a major challenge in oncology, biomedical, and pharmacological research. Despite numerous efforts targeting molecular and pathway-level mechanisms and evaluating combination therapies, current pharmacological approaches remain largely stagnant and show limited consideration of circadian variations in tumor biology and drug resistance. Growing preclinical and clinical evidence suggests that circadian rhythms fundamentally regulate drug..

open-access Research Article

Published: 26 Mar 2026

Association of 12q21.32 cn-LOH Affecting the KITLG p53 Response Element with Therapy Resistance in Acute Leukemia

Volume 2

open-access Review Article

Published: 31 Mar 2026

Here, There, and Everywhere: Alpha-Fetoprotein in Cancer Immunotherapy

Volume 2

Alpha-fetoprotein (AFP) delivers nutrients to immature cells in a shuttle-like manner via alpha-fetoprotein receptor (AFPR)-mediated endocytosis. A small subset of immature myeloid-derived suppressor cells (MDSCs) acts as a key regulator of immune tolerance during pregnancy, cancer, and other conditions. MDSCs, low doses of AFP, and AFP-binding ligands can modulate both the innate and adaptive immune responses. MDSCs decrease excessive immune..